mesangial proliferative glomerulonephritis…

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kidney anatomy, glomerulonephritis, mesangial proliferative, mesangial proliferative gn, mesangial proliferative glomerulonephritis, kidney disorder characterized by swelling, blood, in the urine, dark urine, inflammation of an internal kidney structure, glomerulus, specifically an increase in number of certain glomerular cells, mesangial cells, accompanied by antibody deposits, in the mesangium layer, the glomerular capillary, mesangial proliferative glomerulonephritis, form of glomerulonephritis, inflammation, the kidney glomeruli, mesangial cells, the glomerular capillaries, increase in size, number, giving the glomeruli a lumpy appearance, mechanism that triggers the disorder, believed to be some type of immune response, inflammation, the glomeruli is associated, deposits of antibodies, causes, nephrotic syndrome, protein loss, in the urine, swelling, acute, chronic, rapidly progressive glomerulonephritis, progress to chronic kidney failure, relatively uncommon disorder, term mesangial proliferative gn is actually a description, the microscopic pattern of this disease, seen more commonly in lupus patients who develop gn, in patients, iga mediated kidney disease, iga nephropathy, affect both adults, children, men affected slightly more often than women, swelling, edema, around the eyes, extremities, the feet, ankles, abdomen, general, foamy appearance, the urine, bloody, dark urine, involuntary weight gain, from retention of fluid, poor appetite, high blood pressure, examination is nonspecific, except, edema, signs of acute, chronic renal failure present, high blood pressure, low urine output, fluid overload, urinalysis, reveal blood, variable amounts of protein, fats present, in the urine, bun, serum creatinine elevated, renal failure is present, blood lipids elevated, blood protein levels decreased, renal, kidney, biopsy confirms mesangial proliferative glomerulonephritis, treatment goals, to relieve symptoms, prevent complications, disorder, chronic, requiring prolonged treatment, corticosteroid, immunosuppressive, antihypertensive, diuretic medications given in an attempt to control symptoms, retard progression, the disorder, antibodies indicates there is some type of immune response causing the disorder, suppression, the immune system, corticosteroids, immunosuppressives, not reduce symptoms in all cases, treatment of high blood cholesterol, triglyceride levels recommended to reduce the development of atherosclerosis, dietary limitation of cholesterol, saturated fats of only limited benefit, high levels of cholesterol, triglyceride associated, overproduction by the liver, excessive intake of fats, medications to reduce cholesterol, triglycerides recommended, high-protein diets, debatable value, in many patients, reducing the amount, protein in diet produces a decrease in urine protein, a moderate-protein diet, 1 gram per kilogram of body weight per day, in cases of kidney failure, a low-protein diet is recommended to reduce the accumulation of dangerous nitrogen containing wastes, a by-product of protein metabolism, sodium, salt, fluids restricted to help control swelling, vitamin d, replaced, nephrotic syndrome develops that is unresponsive to therapy, disorder acute, short-term, chronic, unresponsive to therapy, complications, affect the outcome, atherosclerosis, related heart diseases, acute renal failure, chronic renal failure, infections, pneumococcal pneumonia, malnutrition, fluid overload, congestive heart failure, pulmonary edema, symptoms indicating mesangial proliferative glomerulonephritis present, decreased urine output, new symptoms develop.